OpenAlex topic

Kruppel-like factors research

This page lists works and academicians tagged with an OpenAlex topic. It is not a YÖKSİS primary or secondary field.

OpenAlex 601 works 2 author topics

Works

415 works

TMAO-Triggered Endothelial–Mesenchymal Transition and Microvesicle Release as Mediators of Vascular Smooth Muscle Cell Osteogenic Differentiation and Vascular Calcification 2026

Background: Cardiovascular diseases (CVDs) are the leading global cause of mortality, with vascular calcification (VC) as a major predictor of adverse outcomes. Although vascular smooth muscle cells (VSMCs) are established contributors, the role of endothelial cells (ECs), particularly via the endothelial–mesenchymal…
Supplementary Figure S4 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

PRMT1 and DDX3 are associated with poor prognosis.
Supplementary Figure S8 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

PRMT1 is associated with oxidative phosphorylation (OXPHOS) in breast cancer.
Supplementary Figure S9 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

DDX3 depletion induces a metabolic shift to glycolysis.
Supplementary Figure S12 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

PRMT1/DDX3 axis contributes to breast cancer metastasis.
Supplementary Figure S3 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

PRMT1 methylates DDX3.
Supplementary Figure S1 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

Transcriptomic analysis showing that primary and metastatic 4T1 cells participate in distinct pathways.
Supplementary Figure S11 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

PRMT1-mediated DDX3 methylation contributes to recruitment of translation initiating factors, and DDX3 binds to PINK1 mRNA to promotes its translation.
Supplementary Table S1 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

Resource and reagent used in this study.
Supplementary Figure S10 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

Stress induces DDX3 levels and DDX3-mediated mitophagy.
Supplementary Table S2 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

Top 30 Gene Ontology (GO) pathways enriched in 4T1-LM cells compared to 4T1-PT cells.
Supplementary Figure S7 from Arginine Methylation of DDX3 by PRMT1 Mediates Mitochondrial Homeostasis to Promote Breast Cancer Metastasis 2026

DDX3 regulates mitophagy in breast cancer.

Academicians

2 academicians