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Article detail · 2007

NOVEL EVIDENCE SUGGESTING AN ANTI OXIDANT PROPERTY FOR ERYTHROPOIETIN ON VANCOMYCIN INDUCED NEPHROTOXICITY IN A RAT MODEL

YÖKSİS OpenAlex SJR Q2 JCR Q2 Citations 81 Percentile 86.5% FWCI 1.96
Year
2007
Type
article

Data source split

  • YÖKSİS YÖKSİS article record
  • YÖKSİS venue Clinical and Experimental Pharmacology and Physiology
  • Catalog match (ISSN) Clinical and Experimental Pharmacology and Physiology
  • OpenAlex OpenAlex enrichment (abstract, citations, topics)

Abstract

OpenAlex · English

1. The aim of the present study was to investigate the role of oxidative stress in renal injury and to determine whether erythropoietin (EPO) acts as an anti-oxidant in vancomycin (VCM)-induced renal impairment. 2. Twenty-four rats were divided into three groups as follows: (i) control (Group 1); (ii) VCM treated (Group 2); and (iii) VCM + EPO treated (Group 3). Vancomycin (200 mg/kg, i.p.) was administered to Groups 2 and 3 for 7 days. Erythropoietin (150 IU/kg, i.p.) treatment was started 24 h before VCM and lasted for 7 days. On Day 8, renal tissues were excised and blood samples were collected. Serum creatinine and blood urea nitrogen were measured, along with renal malondialdehyde (MDA) levels, superoxide dismutase (SOD) and catalase (CAT) activity and tissue VCM levels. The kidneys were examined for any histopathological changes. 3. Renal MDA levels were found to be increased, whereas SOD and CAT activity was decreased, in the VCM-treated group compared with the control group. There was a marked decrease in MDA levels and an increase in SOD activity, but not CAT activity, after VCM + EPO treatment. Marked histopathological alterations, including interstitial oedema, tubular dilatation, tubular epithelial cell desquamation and vacuolization, were observed in VCM-treated rats. Histopathological changes were significantly improved after EPO administration. 4. In conclusion, the present data suggest that oxidative stress plays an important role in VCM-induced nephrotoxicity. Erythropoietin seems to act as an anti-oxidant, diminishing the toxic oxidative effects of VCM on renal tissues.

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Citations

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81 citations

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Authors

  1. HASAN ÇETİN
  2. OLGAR ŞEREF
  3. FARUK ÖKTEM
  4. İBRAHİM METİN ÇİRİŞ SÜLEYMAN DEMİREL ÜNİVERSİTESİ
  5. EFKAN UZ
  6. ÇAĞATAY ASLAN
  7. MEHMET FEHMİ ÖZGÜNER