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Article detail · 2023

Melatonin: a promising neuroprotective agent for cerebral ischemia-reperfusion injury

Journal

Frontiers Media SA

ISSN 3456

YÖKSİS OpenAlex Open access · gold Citations 32 Top 10% Percentile 93.8% FWCI 3.65
Year
2023
Type
article

Data source split

  • YÖKSİS YÖKSİS article record
  • YÖKSİS venue Frontiers Media SA
  • OpenAlex OpenAlex enrichment (abstract, citations, topics)

Abstract

OpenAlex · English

Cerebral ischemia-reperfusion (CIR) injury is initiated by the generation of reactive oxygen species (ROS), which leads to the oxidation of cellular proteins, DNA, and lipids as an initial event. The reperfusion process impairs critical cascades that support cell survival, including mitochondrial biogenesis and antioxidant enzyme activity. Failure to activate prosurvival signals may result in increased neuronal cell death and exacerbation of CIR damage. Melatonin, a hormone produced naturally in the body, has high concentrations in both the cerebrospinal fluid and the brain. However, melatonin production declines significantly with age, which may contribute to the development of age-related neurological disorders due to reduced levels. By activating various signaling pathways, melatonin can affect multiple aspects of human health due to its diverse range of activities. Therefore, understanding the underlying intracellular and molecular mechanisms is crucial before investigating the neuroprotective effects of melatonin in cerebral ischemia-reperfusion injury.

Topics

Citations

OpenAlex cited_by_count. Not a WoS or Scopus citation count; those sources have no separate column here.

32 citations

OpenAlex cited_by_count (cache / database)

Authors

  1. Majid Tozihi
  2. Behrouz Shademan
  3. Hadi Yousefi
  4. ÇIĞIR BİRAY AVCI EGE ÜNİVERSİTESİ
  5. Alireza Nourazarian
  6. Gholamreza Dehghan