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Article detail · 2018

Mutations in the mitochondrial ribosomal protein MRPS22 lead to primary ovarian insufficiency

Journal

Human Molecular Genetics

ISSN 0964-6906

YÖKSİS OpenAlex Open access · bronze SJR Q1 JCR Q1 Citations 64 Percentile 88.8% FWCI 2.24
Year
2018
Type
article

Data source split

  • YÖKSİS YÖKSİS article record
  • YÖKSİS venue Human Molecular Genetics
  • Catalog match (ISSN) Human Molecular Genetics
  • OpenAlex OpenAlex enrichment (abstract, citations, topics)

Abstract

OpenAlex · English

Primary ovarian insufficiency (POI) is characterized by amenorrhea and loss or dysfunction of ovarian follicles prior to the age of 40. POI has been associated with autosomal recessive mutations in genes involving hormonal signaling and folliculogenesis, however, the genetic etiology of POI most often remains unknown. Here we report MRPS22 homozygous missense variants c.404G>A (p.R135Q) and c.605G>A (p.R202H) identified in four females from two independent consanguineous families as a novel genetic cause of POI in adolescents. Both missense mutations identified in MRPS22 are rare, occurred in highly evolutionarily conserved residues, and are predicted to be deleterious to protein function. In contrast to prior reports of mutations in MRPS22 associated with severe mitochondrial disease, the POI phenotype is far less severe. Consistent with this genotype-phenotype correlation, mitochondrial defects in oxidative phosphorylation or rRNA levels were not detected in fibroblasts derived from the POI patients, suggesting a non-bioenergetic or tissue-specific mitochondrial defect. Furthermore, we demonstrate in a Drosophila model that mRpS22 deficiency specifically in somatic cells of the ovary had no effect on fertility, whereas flies with mRpS22 deficiency specifically in germ cells were infertile and agametic, demonstrating a cell autonomous requirement for mRpS22 in germ cell development. These findings collectively identify that MRPS22, a component of the small mitochondrial ribosome subunit, is critical for ovarian development and may therefore provide insight into the pathophysiology and treatment of ovarian dysfunction.

Topics

Citations

OpenAlex cited_by_count. Not a WoS or Scopus citation count; those sources have no separate column here.

64 citations

OpenAlex cited_by_count (cache / database)

Authors

  1. Anlu Chen
  2. Dov Tiosano
  3. TÜLAY GÜRAN MARMARA ÜNİVERSİTESİ
  4. Hagit N Baris
  5. YAVUZ BAYRAM
  6. Adi Mory
  7. Laura Shapiro-Kulnane
  8. Hodges Craig A
  9. Zeynep C Akdemir
  10. SERAP DEMİRCİOĞLU
  11. Shalini N Jhangiani
  12. den Akker van
  13. Charles L Hoppel
  14. Helen K Salz
  15. James R Lupski
  16. David A Buchner