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VOLKAN SEYRANTEPE

PROFESÖR

İZMİR YÜKSEK TEKNOLOJİ ENSTİTÜSÜ İZMİR YÜKSEK TEKNOLOJİ ENSTİTÜSÜ FEN FAKÜLTESİ

  • Ana Dal Sağlık Bilimleri Temel Alanı
  • Yan Dal Tıbbi Biyoloji

Kayıtlı çıktılara kısa bakış — ayrıntılar aşağıda.

  • Makale 59
  • Proje 0
  • Kitap 0
  • Bildiri 13
  • Patent 0
  • Sanatsal 0
Scopus (SJR) Q1 29 Q2 14 Q3 7 Q4 2
WoS (JCR) Q1 20 Q2 16 Q3 10 Q4 4
TR Index 1 makale

Alan+yıl+tür normalize OpenAlex yüzdelik — Clarivate ESI / SciVal değildir.

Üst %1 makale 0
Üst %10 makale 9
Ort. yüzdelik 71.6%
Üst %1 payı 0.0%
Üst %10 payı 23.1%

Makaleler

YÖKSİS ve OpenAlex kaynak ayrımıyla makaleler; quartile ve TR Index ile daraltabilirsiniz.

Dizin filtreleri

Toplam 59 yayın

Scopus (SJR)
Q1 29 Q2 14 Q3 7 Q4 2
WoS (JCR)
Q1 20 Q2 16 Q3 10 Q4 4
TR Index
1 makale

Makale listesi

  1. 2026 Intrathecal delivery of AAVrh10-mHexa combined with anti-inflammatory treatment reduces neuropathological markers and extends the lifespan of mice with early-onset Tay-Sachs disease METABOLIC BRAIN DISEASE DOI 10.1007/s11011-026-01802-2 YÖKSİS SJR Q2 JCR Q2 OpenAlex 15.5%
  2. 2025 A Comprehensive microRNA-seq Transcriptomic Analysis of Tay-Sachs Disease Mice Revealed Distinct miRNA Profiles in Neuroglial Cells JOURNAL OF MOLECULAR NEUROSCIENCE DOI 10.1007/s12031-025-02395-8 YÖKSİS SJR Q2 JCR Q3 OpenAlex 67.9%
  3. 2025 Imbalance in redox homeostasis is associated with neurodegeneration in the murine model of Tay-Sachs disease. Molecular biology reports DOI 10.1007/s11033-025-10380-y YÖKSİS SJR Q2 JCR Q3 OpenAlex 86.9%
  4. 2025 Combined treatment of Ketogenic diet and propagermanium reduces neuroinflammation in Tay-Sachs disease mouse model. Metabolic brain disease DOI 10.1007/s11011-025-01553-6 YÖKSİS SJR Q2 JCR Q2 OpenAlex 89.9%
  5. 2025 Therapeutic targeting of neuroinflammation in sphingolipidosis Molecular Immunology DOI 10.1016/j.molimm.2025.09.005 YÖKSİS SJR Q2 JCR Q2 OpenAlex 14.4%
  6. 2025 Yeast-Produced Human Recombinant Lysosomal β-Hexosaminidase Efficiently Rescues GM2 Ganglioside Accumulation in Tay-Sachs Disease Journal of personalized medicine DOI 10.3390/jpm15050196 YÖKSİS SJR Q1 JCR Q1 OpenAlex 65.0%
  7. 2025 Mass spectrometric profiling reveals alterations in N-Glycans and O-Glycans in Tay-Sachs disease under Autophagy-Induced conditions Glycoconjugate Journal DOI 10.1007/s10719-025-10203-z YÖKSİS SJR Q2 JCR Q3 OpenAlex 54.5%
  8. 2024 Abnormally accumulated GM2 ganglioside contributes to skeletal deformity in Tay-Sachs mice Journal of Molecular Medicine DOI 10.1007/s00109-024-02498-6 YÖKSİS SJR Q1 JCR Q1 OpenAlex 66.5%
  9. 2024 Silencing of B4Galnt1 Gene Prevents GM2 Accumulation in Tay-Sachs Cells Advances in Cell and Gene Therapy DOI 10.1155/2024/1099113 YÖKSİS OpenAlex 51.6%
  10. 2024 Induction of Exocytosis Rescues Lysosomal GM2 Accumulation in Tay-Sachs Disease Advances in Cell and Gene Therapy DOI 10.1155/2024/4047025 YÖKSİS OpenAlex 60.5%
  11. 2024 Lithium treatment rescues dysfunctional autophagy in the cell models of Tay-Sachs disease. Molecular genetics and metabolism DOI 10.1016/j.ymgme.2024.108140 YÖKSİS SJR Q2 JCR Q2 OpenAlex 59.6%
  12. 2024 Gangliosides as Therapeutic Targets for Neurodegenerative Diseases JOURNAL OF LIPIDS DOI 10.1155/2024/4530255 YÖKSİS SJR Q4 JCR Q1 OpenAlex 85.1%
  13. 2023 Autophagic flux is impaired in the brain tissue of Tay-Sachs disease mouse model PLoS One DOI 10.1371/journal.pone.0280650. YÖKSİS SJR Q1 JCR Q1 OpenAlex 78.3%
  14. 2023 Molecular Trojan Horses for treating lysosomal storage diseases Elsevier BV DOI 10.1016/j.ymgme.2023.107648 YÖKSİS SJR Q1 JCR Q2 OpenAlex 67.1%
  15. 2022 Analysis of Brain Lipids in the Early-Onset Tay–Sachs Disease Mouse Model With the Combined Deficiency of β-Hexosaminidase A and Neuraminidase 3 Frontiers in Molecular Biosciences DOI 10.3389/fmolb.2022.892248 YÖKSİS SJR Q1 JCR Q2 OpenAlex 66.6%
  16. 2021 Sialidase neu4 deficiency is associated with neuroinflammation in mice Glycoconjugate Journal DOI 10.1007/s10719-021-10017-9 YÖKSİS SJR Q3 JCR Q3 OpenAlex 53.1%
  17. 2020 GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs disease Journal of Neuroinflammation DOI 10.1186/s12974-020-01947-6 YÖKSİS OpenAlex üst %10 OpenAlex 90.3%
  18. 2018 Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease Experimental Neurology DOI 10.1016/j.expneurol.2017.09.012 YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %10 OpenAlex 93.0%
  19. 2018 Identification of cytoplasmic sialidase NEU2-associated proteins by LC-MS/MS Turkish Journal of Biochemistry-Turk Biyokimya Dergisi DOI 10.1515/tjb-2018-0089 YÖKSİS SJR Q3 JCR Q4 OpenAlex 44.4%
  20. 2018 The Second Case of Saposin A Deficiency and Altered Autophagy JIMD Reports DOI 10.1007/8904_2018_114 YÖKSİS SJR Q3 OpenAlex üst %10 OpenAlex 91.8%

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