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Akademisyen

HAKAN KÜÇÜKSAYAN

DOKTOR ÖĞRETİM ÜYESİ

KASTAMONU ÜNİVERSİTESİ TIP FAKÜLTESİ TEMEL TIP BİLİMLERİ BÖLÜMÜ

  • Ana Dal Sağlık Bilimleri Temel Alanı
  • Yan Dal Tıbbi Biyoloji

Kayıtlı çıktılara kısa bakış — ayrıntılar aşağıda.

  • Makale 12
  • Proje 0
  • Kitap 3
  • Bildiri 18
  • Patent 0
  • Sanatsal 0
Scopus (SJR) Q1 3 Q2 1 Q3 4 Q4 0
WoS (JCR) Q1 1 Q2 2 Q3 3 Q4 2
TR Index 0 makale

Alan+yıl+tür normalize OpenAlex yüzdelik — Clarivate ESI / SciVal değildir.

Üst %1 makale 0
Üst %10 makale 1
Ort. yüzdelik 55.4%
Üst %1 payı 0.0%
Üst %10 payı 8.3%

Makaleler

YÖKSİS ve OpenAlex kaynak ayrımıyla makaleler; quartile ve TR Index ile daraltabilirsiniz.

Dizin filtreleri

Toplam 12 yayın

Scopus (SJR)
TR Index
0 makale

Makale listesi

  1. 2024 Elevated level of neuroserpin is an indication for the resistance to gambogic acid-induced apoptosis and oxidative stress in triple-negative breast cancer cells Asian Biomedicine DOI 10.2478/abm-2024-0010 YÖKSİS SJR Q3 JCR Q4 OpenAlex 83.7%
  2. 2022 The dicyano compound induces autophagic or apoptotic cell death via Twist/c-Myc axis depending on metastatic characteristics of breast cancer cells Molecular Biology Reports DOI 10.1007/s11033-021-06817-9 YÖKSİS SJR Q2 JCR Q3 OpenAlex 65.9%
  3. 2021 Transforming Growth Factor-Beta (TGF-β) Employs Epithelial-Mesenchymal Transition (EMT)-Inducible Cancer Cells to Secrete the Exosomal miRNAs Regulating the Immune Response International Journal of Academic Medicine and Pharmacy DOI 10.29228/jamp.51913 YÖKSİS OpenAlex 7.1%
  4. 2019 NF-κB-Induced Upregulation of miR-548as-3p Increases Invasion of NSCLC by Targeting PTEN Bentham Science Publishers Ltd. DOI 10.2174/1871520619666190206165215 YÖKSİS SJR Q3 JCR Q3 OpenAlex 57.2%
  5. 2017 The crosstalk between p38 and Akt signaling pathways orchestrates EMT by regulating SATB2 expression in NSCLC cells Tumor Biology DOI 10.1177/1010428317706212 YÖKSİS SJR Q1 JCR Q2 OpenAlex 70.3%
  6. 2016 Downregulation of SATB2 is critical for induction of epithelial-to-mesenchymal transition and invasion of NSCLC cells Lung Cancer DOI 10.1016/j.lungcan.2016.05.032 YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %10 OpenAlex 90.1%
  7. 2015 Akciğer kanseri hücre dizilerinde hipoksi indüklenebilir faktör-1 (HIF-1) ve paraoksonaz enzim ilişkisinin araştırılması Dicle Medical Journal/Dicle Tip Dergisi DOI 10.5798/diclemedj.0921.2015.03.0590 YÖKSİS OpenAlex 7.3%
  8. 2024 Elevated level of neuroserpin is an indication for the resistance to gambogic acid-induced apoptosis and oxidative stress in triple-negative breast cancer cells Asian Biomedicine DOI 10.2478/abm-2024-0010 YÖKSİS SJR Q3 JCR Q4 OpenAlex 83.6%
  9. 2021 Transforming Growth Factor-Beta (TGF-β) Employs Epithelial-Mesenchymal Transition (EMT)-Inducible Cancer Cells to Secrete the Exosomal miRNAs Regulating the Immune Response International Journal of Academic Medicine and Pharmacy DOI 10.29228/jamp.51913 YÖKSİS OpenAlex 7.1%
  10. 2019 NF-κB-Induced Upregulation of miR-548as-3p Increases Invasion of NSCLC by Targeting PTEN Anti-Cancer Agents in Medicinal Chemistry DOI 10.2174/1871520619666190206165215 YÖKSİS SJR Q3 JCR Q3 OpenAlex 57.2%
  11. 2018 TGF-β–SMAD–miR-520e axis regulates NSCLC metastasis through a TGFBR2-mediated negative-feedback loop Carcinogenesis DOI 10.1093/carcin/bgy166 YÖKSİS SJR Q1 JCR Q2 OpenAlex 67.8%
  12. 2016 Pl3K/Akt/NF-κB Signalling Pathway on NSCLC Invasion Medicinal Chemistry DOI 10.4172/2161-0444.1000351 YÖKSİS OpenAlex 67.7%

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