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OpenAlex konusu

Prion Diseases and Protein Misfolding

Bu sayfa OpenAlex konu etiketine göre çalışmaları ve o konuda görünen akademisyenleri listeler. YÖKSİS temel alan / yan dal değildir.

OpenAlex 423 eser 5 yazar konusu

Çalışmalar

423 eser

  1. Global, regional, and national burden of neurological disorders, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016 2019
    YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %1 OpenAlex 100.0%

    BACKGROUND: Neurological disorders are increasingly recognised as major causes of death and disability worldwide. The aim of this analysis from the Global Burden of Diseases, Injuries, and Risk Factors Study (GBD) 2016 is to provide the most comprehensive and up-to-date estimates of the global, regional, and national…

  2. Neurobrucellosis Clinical and Diagnostic Features 2013
    YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %1 OpenAlex 99.7%

    BACKGROUND: We describe the neurological involvement in brucellosis and revisited diagnostic criteria for neurobrucellosis. METHODS: Patients with laboratory-confirmed brucellosis who were consequently hospitalized were observed prospectively in a brucellosis-endemic region. The neurobrucellosis was diagnosed by any o…

  3. Neurobrucellosis: Clinical and Diagnostic Features 2013
    YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %1 OpenAlex 99.7%

    BACKGROUND: We describe the neurological involvement in brucellosis and revisited diagnostic criteria for neurobrucellosis. METHODS: Patients with laboratory-confirmed brucellosis who were consequently hospitalized were observed prospectively in a brucellosis-endemic region. The neurobrucellosis was diagnosed by any o…

  4. Molecular Mechanisms of Early and Late LTP 2019
    YÖKSİS SJR Q1 JCR Q3 OpenAlex üst %10 OpenAlex 95.3%

    Özet henüz yok.

  5. Aggravation of ischemic brain injury by prion protein deficiency: Role of ERK-1/-2 and STAT-1 2005
    OpenAlex üst %10 OpenAlex 95.2%

    Özet henüz yok.

  6. Neurofilament ELISA validation 2010
    YÖKSİS SJR Q2 JCR Q3 OpenAlex 83.4%

    Özet henüz yok.

  7. Neurofilament ELISA validation 2010
    YÖKSİS SJR Q2 JCR Q3 OpenAlex 83.9%

    Özet henüz yok.

  8. Neurofilament ELISA validation 2010
    YÖKSİS SJR Q2 JCR Q3 OpenAlex 83.6%

    Özet henüz yok.

  9. Hyperphosphorylated tau and paired helical filament like structures in the brains of mice carrying mutant amyloid precursor protein and mutant presenilin 1 transgenes 2003
    YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %10 OpenAlex 92.2%

    Özet henüz yok.

  10. Neurodegenerative Changes Associated with β-Amyloid Deposition in the Brains of Mice Carrying Mutant Amyloid Precursor Protein and Mutant Presenilin-1 Transgenes 2001
    OpenAlex üst %10 OpenAlex 90.8%

    Özet henüz yok.

  11. In Silico Cross Seeding of A and Amylin Fibril like Oligomers 2013
    YÖKSİS SJR Q1 JCR Q1 OpenAlex üst %10 OpenAlex 94.8%

    Recent epidemiological data have shown that patients suffering from Type 2 Diabetes Mellitus have an increased risk to develop Alzheimer's disease and vice versa. A possible explanation is the cross-sequence interaction between Aβ and amylin. Because the resulting amyloid oligomers are difficult to probe in experiment…

  12. Insights into the Molecular Mechanisms of Alzheimer’s and Parkinson’s Diseases with Molecular Simulations: Understanding the Roles of Artificial and Pathological Missense Mutations in Intrinsically Disordered Proteins Related to Pathology 2018
    YÖKSİS SJR Q1 JCR Q2 OpenAlex üst %10 OpenAlex 95.9%

    Amyloid-β and α-synuclein are intrinsically disordered proteins (IDPs), which are at the center of Alzheimer's and Parkinson's disease pathologies, respectively. These IDPs are extremely flexible and do not adopt stable structures. Furthermore, both amyloid-β and α-synuclein can form toxic oligomers, amyloid fibrils a…

Akademisyenler

5 akademisyen